This disease is very common especially in old age, it begins to manifest with occasional forgetfulness that over time become more frequent and unconscious speech. It is a bad progressive and irreversible, if recognized early symptoms can be treated, improving the quality of life of patients.

This disease has three stages:

You can not specify exactly when triggered, but progressive forgetfulness begins with things and situations, confusion about directions, decisions and money management. The patient is spontaneous does not own initiative, it is repetitive in their actions, the mood suddenly changes, becomes disoriented in time and space and began to suffer difficulties in language.

In the second stage, the disease is aggravated and start to ignore friends and family, not held in memory of new experiences, wanders, comes and goes without knowing it, have anxiety, muscle spasms, difficulty expressing their thoughts, and often gets edgy and nervous. Also becomes careless, confused, regarding himself and his health, can not live alone, cooking, cleaning, or shopping, is losing even in his own home, start seeing or hearing things that do not exist and appears incontinence.

In the third stage, late in the disease, the patient is very dependent on the caregiver, memory problems are really serious, and it becomes obvious physical deterioration. Lose weight despite a good diet, can not perform any daily activity, communicates little or nothing from their loved ones, loses the ability to walk, sleep enough, and is likely to get all kinds of infections, especially respiratory.

A simple eye test could detect Alzheimer’s disease and other disorders before symptoms appear, say British scientists.

The dying cells appear as green dots in the retina of ration. Its technique uses fluorescent markers that attach to the dying cells. They are a first indication of cell death in the brain and can be seen in the retina.

The investigation is being conducted in mice but is planning human clinical trials, say researchers from the University of London.

The aim, they say, is that this work will lead to a test available in ophthalmology clinics to spot the disease among the general public.

The research, published in the journal Cell Death and Disease (Cell Death and Disease), could help scientists overcome the difficulties currently facing to investigate what is happening inside the brain of Alzheimer’s sufferers.

Alzheimer’s disease is the most common cause of dementia among older people found 65. It is estimated that one in 20 people between 50 and 70 years suffer from this disease. For those older than 85, the number affected is 20 percent, and it is estimated that this figure will increase due to increased life expectancy in developed countries.

It is a slowly progressive disease, characterized by a progressive dementia with memory loss, decreased ability to perform routine tasks, difficulties in trials, disorientation, personality changes and loss of skills language.

At first, the person has difficulty learning and retaining new information. Then she begins to lose memory and has trouble remembering people and events of childhood or youth. Then other symptoms, such as aphasia, which is the difficulty in expressing thoughts through language.

Another symptom is what is called agnosia, which is difficulty interpreting familiar faces or objects known. The patient can not manage money or remember to take medicine, for example. It also tends to lose the sense of direction and ignores how to get home in case of going for a walk. Anyway, in the early stages of the disease may be feeding, bathing and dressing without help. But the personality changes, irritability, anxiety or depression, can cause serious problems in relationships with family and friends.

In more advanced stages, patients may experience hallucinations and irrational thoughts, and believe they are persecuted or that someone wants to steal their belongings.

A study led by Mount Sinai School of Medicine in New York (USA) found in mice, a link between a diet high in protein and Alzheimer’s disease. The findings are published in the journal Molecular Neurodegeneration.

The researchers evaluated four different menus in a transgenic mouse model of Alzheimer’s disease, which were fed a normal diet, a diet high in fat, low carbohydrate, high protein diet, low carbohydrate or a high option carbohydrates and low in fat.

Then the researchers examined the brain and body weight of mice and the accumulation of plaques and the differences in the structure of several brain regions involved in memory problems that occur in Alzheimer’s disease.

The mice were fed a diet high in protein, low carbohydrate had brains five percent lighter than the other and the regions of hippocampus were less developed. The result was a surprise to researchers and until proved this effect in non-transgenic mice is not clear whether brain mass loss associated with Alzheimer’s plaques.

However, the results of recent studies led the authors to consider a possible theory that high protein diets could leave neurons more vulnerable to Alzheimer’s plaques. The mice ate a high fat diet had higher levels of plaque proteins but this had no effect on plaque burden.

According says Sam Gandy, director of the study, “given the well-known association of high protein diet with the neurotoxicity associated with age, one wonders whether particular diets, when consumed at certain ages, may increase susceptibility to incidence or progression of Alzheimer’s disease.

The researchers note that the only way to answer this approach is to conduct more clinical trials on diet. “The project would be a challenge but might be worth if there were any probability that the ravages of Alzheimer’s may slow down or avoided through a healthy diet. These trials will be needed if scientists want to make specific recommendations about the risks of the diet in Alzheimer’s.